Quinapril
CHICAGO--Cardiovascular disease is associated with endothelial dysfunction, which is caused mainly by the production of radical oxygen species that can destroy nitric oxide NO ; and impair its beneficial vascular effects. Restoration of endothelial function in individuals with hypertension has been associated with improved event-free survival.1 "Thus, the restoration of endotheliumdependent vasodilation is increasingly recognized as an appropriate adjunctive goal of cardiovascular treatment, " said Stefano Taddei, MD. Endothelial function can be restored through nonpharmacologic treatment. Perhaps the most effective nonpharmacologic intervention is physical exercise, which restores NO availability.2, 3 Drug therapy is also effective. The endothelial effect of antihypertensive drugs in several drug classes has been evaluated. Dr Taddei briefly reviewed the published literature for beta-blockers, diuretics, renin-angiotensin-aldosterone system RAAS ; blockers, and calcium antagonists. arm blood flow. The endothelial effects of carvedilol have been evaluated in 1 published report; Matsuda and colleagues5 found that carvedilol significantly improved flow-mediated dilation in patients with coronary artery disease CAD ; . cial effect on endothelial function, " noted Dr Taddei. Within the ACE inhibitor class, Schiffrin and Deng8 found that cilazapril therapy significantly improved endothelium-dependent vasodilation in arterioles of patients with hypertension. Perindopril has been shown to increase flow-mediated dilation, reduce oxidative stress, and increase plasma antioxidant capacity in patients with hypertension.9 Ramipril therapy caused dose-dependent vasodilation in patients with hypertension in a study by Ghiadoni and colleagues10; investigators found that although both 5-mg day and 10mg day doses were effective, the 10mg day dose induced higher NO levels and a greater improvement in NO-dependent vasodilation compared with 5 mg day. Furthermore, ramipril has been shown to benefit endothelial function in patients with CAD. In a study of patients with CAD, Hornig and colleagues11 reported that 4 weeks of therapy with ramipril increased NO bioavailability and improved endothelial function. Qiunapril has also demonstrated an endothelial benefit in patients with CAD. In the Trial on Reversing ENdothelial Dysfunction TREND ; , 12 quinapril improved endothelial vasomotor function in patients with CAD. "It is important to note that TREND patients were normotensive and did not have pronounced dyslipidemia or evidence of heart failure, " said Dr Taddei. "The benefits of quinapril treatment were probably due to a reduction of the deleterious effects of angiotensin II and to enhancement of the beneficial effects of endothelial NO release." Several ARBs have demonstrated beneficial effects on human endothelial function. In their study of patients with hypertension, Klingbeil and colleagues6 found that valsartan improved NO production and release. Ghiadoni and colleagues13 demonstrated that candesartan improved NO release and reduced vasoconstriction to endogenous endothelin-1 in patients with hypertension. Schiffrin and colleagues14 discovered that treatment with losartan restored endothelial function in resistance arteries of patients with essential hypertension. Moreover, losartan has been shown to increase NO bioavailability and improve endothelial function in patients with CAD, 11 and to reverse abnormal coronary vasomotion in patients with atherosclerosis or its risk factors.15.
Increasing evidence supports an association between inflammation and plaque rupture. Macrophages and vascular smooth muscle cells are a source of cytokines and growth factors , which contribute to ongoing inflammation during atherogenesis. In a rabbit model of atherosclerosis , we evaluated the effect of the ACE inhibitor quinapril on different parameters implicated in the pathogenesis of the plaque , such as the presence of chemokines interleukin-8 , monocyte chemoattractant protein-1 ; , collagen I , and vascular smooth muscle cell proliferation PDGF-B ; . Since nuclear factor B NF- B ; has been implicated in the control of chemokine transcription and cell proliferation , we also investigated its activation and localization in the lesion. Uqinapril administration for 28 days caused a down-regulation in arterial expression of interleukin-8 and monocyte chemoattractant protein-1 mRNA and protein ; . However , collagen I expression mRNA and protein ; was not modified. PDGF-B expression was reduced in both the intima and the media. Active NF- B , found in both macrophages and vascular smooth muscle cells , was also reduced by quinapril. Nevertheless , no significant changes were noted in the mild neointima formation, although a certain trend toward normalization was found in the quinapril-treated group. In conclusion, our results show that quinapril treatment attenuates several parameters associated with inflammation within the atherosclerotic lesions that are controlled by NF- B , although it has no effect on collagen I expression. Both effects could contribute to the stabilization of the atherosclerotic plaque. J Pathol 1998, 153: 18251837.
Quinapril 5mg
PREMPRO. 28 prenatal vitamins. 35 PREVACID . 32 PREVACID inj. 32 PREVPAC. 32 PREZISTA. 11 PRILOSEC 40 mg. 32 primidone. 20 PROAIR HFA . 36 probenecid . 7 procainamide 250 mg, 500 mg. 17 PROCAINAMIDE 750 mg, 1000 mg . 17 PROCANBID . 17 prochlorperazine. 30 prochlorperazine inj . 30 PROCRIT. 33 PROGLYCEM . 28 PROGRAF. 34 PROLEUKIN. 14 promethazine. 30 promethazine inj . 30 propafenone . 17 propranolol. 18 propranolol ext-rel. 18 propranolol inj . 18 propylthiouracil. 29 PROSTIGMIN. 24 PROTOPIC . 40 PROVENTIL HFA . 36 PROVIGIL . 24 PSORCON E crm, oint 0.05% . 40 PULMOZYME . 38 pyrazinamide . 11 pyridostigmine inj . 24 pyridostigmine tabs . 24 QUALAQUIN . 10 quinapril . 16 quinidine gluconate ext-rel 324 mg. 17 quinidine sulfate 200 mg, 300 mg . 17 quinidine sulfate ext-rel 300 mg . 17 QVAR . 37 RABIES VACCINE . 35 RANEXA . 19 ranitidine . 31 ranitidine inj. 31 RAPAMUNE. 34 RAPTIVA. 39 RAZADYNE. 20 RAZADYNE ER. 20 Page 52!
Novolin, novolog, insulin novolin ge toronto delivery with a meal, for example, quinapril brand.
Benazepril Lotensin ; captopril Capoten ; enalapril Vasotec ; enalaprilat Vasotec I.V. ; fosinopril Monopril ; lisinopril Prinival, Zestril ; moexipril Univasc ; quinapril Accupril ; ramipril Altace ; trandolapril Mavik.
Potential mechanism. Theoretically, two mechanisms can be involved. Triglyceride-rich lipoproteins may, in analogy to LDL, induce increased oxygen radical production by a reninangiotensin modifiable pathway. Membrane oxidases, which have been implicated as important superoxideproducing enzymes in the vasculature, are in part ; regulated by angiotensin II 14 ; . Hence, decreased angiotensin II formation may result in down-regulation of the expression of membrane oxidases, resulting in decreased lipid-induced superoxide production. The ensuing increase in NO bioavailability can arise either due to decreased destruction of NO by decreased superoxide itself or due to decreased formation of, for example, oxidatively modified chylomicrons, which may interfere with NO production and or NO bioavailability. In addition, bradykinin-mediated stimulation of endothelial function may also be involved, which is supported by the significant difference in effect between quinapril and losartan in preventing the change in postprandial endothelial function. In agreement, the protective effect of ACE inhibition against the toxicity of ox-LDL in rat aortic rings was also shown to be bradykinin dependent 23 ; . Interestingly, preprandial endothelial dysfunction was unaffected by ACE inhibition, which may imply that the bradykinin pathway is of particular importance during dyslipidemia. In this respect, bradykinin-stimulated endothelial function has been shown to be preserved in hypercholesterolemic patients, whereas other agonist-stimulated endothelial function, that is, acetylcholine, serotonin and alpha2 receptor stimulation, is impaired 28 ; . Alternatively, differences in pharmacokinetics, such as binding and tissue penetration, may have contributed to the observed differences between quinapril and losartan. It is less likely that the difference in effect is the result of the dosage of losartan used in this study, since losartan reaches a plateau in the doseresponse curve after 50 mg 29 ; . The exact nature of the interaction between lipid-induced endothelial dysfunction and the reninangiotensin system deserves further research. Nevertheless, the observation that postprandial endothelial dysfunction can be improved by blockade of the reninangiotensin system provides a rationale for ACE inhibition and or angiotensin type 1 receptor antagonist therapy in patients with impaired remnant particle clearance such as diabetes and familial combined hyperlipidemia and aceon.
Effects of angiotensin-converting enzyme inhibition on transient ischemia: The quinapril anti-ischemia and symptoms of angina reduction QUASAR ; trial Carl J. Pepine, Jean-Lucien Rouleau, Karen Annis, Anique Ducharme, Patrick Ma, Jacques Lenis, Richard Davies, Udho Thadani, Bernard Chaitman, Harry E. Haber, S. Ben Freedman, Milton L. Pressler, Bertram Pitt, and QUASAR Study Group J. Am. Coll. Cardiol. 2003; 42; 2049-2059 doi: 10.1016 j.jacc.2003.07.027.
Farewells Over the past year we have bid farewell to the following fellows. Jim Watterson has accepted a position in endourology at the Ottawa Hospital. Mario Sofer is currently practicing Endourololgy at the University of Tel Aviv in Israel after spending a year in Miami following his fellowship with Dr. Denstedt. Wei Zheng is a community urologist in California. Jeffrey Zhang returned to China after a one-year research fellowship in uro-oncology sponsored in part by the Societe Internationale d'Urologie. He has taken up an academic position in Chang-Sha University, China. Toru Onita has returned to Nagasaki University in Japan after completing his fellowship in oncology. James Tan has retuned to an academic position in Singapore after completing his fellowship in uro-oncology here. Anand Khakar completed his renal transplantation fellowship but before returning to a University faculty position in Chennai, India, he is staying on for one year as a multiorgan transplant fellow. Darren Beiko from Queen's University is completing his 2 nd year endourology fellowship. Bodo Knudsen from University of Manitoba is completing his first year research ; endourology fellowship. He is looking forward to his clinical fellowship year starting July 2003. Arrivals In July 2003, we will welcome Ben Chew from the University of Toronto and Andrew Tan from New Zealand to the Endourology Fellowship program. Andrew Tan is the recipient of the CIRCON-ACMI Endourology Fellowship. NEW: Uro-Oncology Fellowship Program Aventis Pharma has announced a $50, 000 donation, representing one year of funding, in support of a UroOncology Fellowship position. This is an integrated fellowship, involving medical oncology, urology and radiation oncology. The fellowship is a two-year multidisciplinary integrated programme, with one clinical year and one research year. Participating institutions in this programme include LHSC, UWO and LRCC. The Uro-Oncology fellowship position will be an integral component of the London Prostate Cancer Centre and perindopril, because quinapril side effects.
This is a pretty rich offering, and one that the present medical community has no qualms about discarding.
No significant changes in digoxin pharmacokinetics have been reported with benazepril, enalapril, lisinopril, moexipril, perindopril, quinapril, ramipril, or trandolapril and sumycin.
Quinapril contraindications
Mg123 day, given as a single dose or in two equally divided doses. In some patients treated once daily, the antihypertensive effect may diminish toward the end of the dosing interval. In such patients an increase in dosage or twice daily administration may be warranted. In general, doses of 4080 mg and divided doses give a somewhat greater effect at the end of the dosing interval. Concomitant Diuretics: If blood pressure is not adequately controlled with ACCUPRIL monotherapy, a diuretic may be added. In patients who are currently being treated with a diuretic, symptomatic hypotension occasionally can occur following the initial dose of ACCUPRIL. To reduce the likelihood of hypotension, the diuretic should, if possible, be discontinued 2 to 3 days prior to beginning therapy with ACCUPRIL see WARNINGS ; . Then, if blood pressure is not controlled with ACCUPRIL alone, diuretic therapy should be resumed. If the diuretic cannot be discontinued, an initial dose of 5 mg ACCUPRIL should be used with careful medical supervision for several hours and until blood pressure has stabilized. The dosage should subsequently be titrated as described above ; to the optimal response see WARNINGS, PRECAUTIONS, and Drug Interactions ; . Renal Impairment: Kinetic data indicate that the apparent elimination half-life of quinaprilat increases as creatinine clearance decreases. Recommended starting doses, based on clinical and pharmacokinetic data from patients with renal impairment, are as follows: Creatinine Clearance 60 mL min 3060 mL min 1030 mL min 10 mL min Maximum Recommended Initial Dose 10 mg 5 mg 2.5 mg Insufficient data for dosage recommendation.
Indirect Economic Benefits Lilly's ability to attract the most talented employees, tap scientific insights, develop effective supplier relationships, and make sound investment decisions all depends to a large extent on conditions in our surrounding communities. Fortunately, Lilly's headquarters and our operations worldwide are located in exceptional communities--places that combine aesthetic and cultural appeal, attractive and stable economic conditions, and a critical mass of related businesses. We benefit from these communities--and it is natural that we should give back much in return and risedronate.
Before taking ramipril, tell your doctor and pharmacist if you are allergic to ramipril, benazepril lotensin ; , captopril capoten ; , enalapril vasotec ; , fosinopril monopril ; , lisinopril prinivil, zestril ; , moexipril univasc ; , perindopril aceon ; , quinnapril accupril ; , trandolapril mavik ; , or any other medications.
Doctors measured using ultraviolet light. The Journal of Skin Pharmacology and Physiology will also publish new findings later in 2007 from the University of Naples, Italy, also applied a treatment to the surface of the skin topical ; that contained 50 parts-per-million ppm ; lutein and 2 ppm zeaxanthin per day, supplying 50 mcg of lutein and 2 mcg of zeaxanthin per gram of topical treatment. Every two weeks for 12 weeks, researchers measured skin moisture hydration ; , ability to maintain size and shape elasticity ; , protective fat layer superficial skin lipids ; , and cell membrane damage lipid peroxidation ; , and found that, compared to placebo, those who had taken lutein and zeaxanthin orally, topically, alone or together, had significantly improved in each measure. The oral-topical combination group averaged 60% greater hydration, 20% better elasticity, 50% more lipid protection, and 64% less cell damage and salmeterol.
We have only just begun to analyze and medically evaluate compounds from our rainforests systematically, because side effects of quinapril.
Treating the underlying inflammation of the airways is essential for preventing and maintaining control of asthma symptoms. When do you start daily therapy with a long-term control medication? and fluticasone.
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Each of these medications will be discussed in detail in the articles below, for example, qquinapril 5 mg.
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The word asthma comes from the ancient Greek word for `panting attack'. There are many theories about the possible causes of asthma environment changes, diet, or exposure to different infections. owever, we don't know for sure why ew ealand has one of the highest asthma rates in the world. We can't guard ourselves against getting asthma, but we can control how much it affects our life. Work with your doctor, nurse, pharmacist and asthma educator to learn as much as you can about your asthma. It feels great to look after your asthma and be able to lead the life you want.
HCC ; . Conversely, the presence of genetically damaged or variant estrogen receptors vERs ; seems to correlate with increased risk of HCC. Nonetheless, the role of estrogens in the development of HCC remains somewhat controversial and is an area of active research. Alcohol, Estrogen, and the Liver: A Bad Mix While it appears estrogen may offer some protection to women with non-alcoholic liver disease, the effects of chronic alcohol use are more severe in women than in men. Compared to men, women develop alcoholinduced liver disease over a shorter period of time and with less consumption of alcohol. Women are also more likely than men are to develop alcoholic hepatitis and die from alcohol-related cirrhosis. Research in animal models suggests women's increased susceptibility to the liver damaging effects of alcohol is related to estrogen-mediated pathways. Hepatitis and Pregnancy Although women with cirrhosis can have impaired fertility, women with less severe liver disease generally retain their fertility. This is important for several reasons. First and foremost, it is important for all women to know their fertility status in order to have the desired control over their reproductive choices. Many women with chronic hepatitis are able to safely conceive and bear healthy children. There appears to be no increased incidence of pregnancy or birth-related complications among women with chronic, non-cirrhotic hepatitis. However, women with chronic hepatitis B or C should work closely with their health care providers to minimize the risk of transmission of the virus to the fetus. The risk of maternal-fetal transmission is around 5% for women with hepatitis C and 10-20% for most women with hepatitis B and albenza and quinapril, because side effect.
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Warnings precautions do not stop using this drug without first consulting your doctor and albendazole.
Fig. 9. Angiotensin-converting enzyme ACE ; activity in luteal cells. Rat luteal cells were incubated for 24 h in the absence or presence of LH 100 ng ml ; . Subsequently, ACE activity was determined using as described in METHODS. Data are corrected for substrate blanks and are expressed as percent fluorescence of control incubations. Quinaprl 100 M ; was included where indicated. Values are means SE n 4 ; experiment. a, bP 0.05, significantly different means.
FIG. 3. Comparative alignment of partial 16S rRNA gene sequences of Brucella canis, Rasbo bacterium, and amplicons derived from dogs 4 to 10. The sequence of the noncoding RNA strand is shown. The numbers correspond to the Escherichia coli numbering scheme. Unless noted in boldface type in the table, all intervening sequences between no. 45 and 207 for all isolates were identical. Dashes a ; indicate the lack of comparable sequence. aP, alpha proteobacterium.
Castration, hormonal regulation, mesentery blood flow, neuronal nitric oxide synthase, protein kinase C, 821 catheter infection, bacterial endocarditis, hemodialysis, intravenous catheter, 563 CD4 antigen, Human immunodeficiency virus infection, kidney disease, proteinuria, 516 cefazolin, ceftazidime, continuous ambulatory peritoneal dialysis, 569 cefotaxime, ciprofloxacin, imipenem, recurrent infection, Salmonella enterica, salmonellosis, 590 ceftazidime, cefazolin, continuous ambulatory peritoneal dialysis, 569 cell cycle G2 phase, prostate cancer, radiosensitivity, 769 cell growth, alpha smooth muscle actin, bladder muscle, bone marrow cell, collagen, intestine mucosa, tissue engineering, 696 cell interaction, bone metastasis, prostate cancer, 746 cellular immunity, chronic kidney failure, continuous ambulatory peritoneal dialysis, innate immunity, 584 chaperone, B lymphocyte, galactosyltransferase, immunoglobulin A nephropathy, peripheral lymphocyte, 476 childhood disease, bladder stone, bladder surgery, endemic disease, lithotomy, 689 - kidney disease, oxidative stress, 543 - nephrolithiasis, 505 chlamydiasis, 819 4 [5 4 chlorophenyl ; 3 trifluoromethyl 1h pyrazol 1 yl]benzenesulfonamide, kidney injury, renal protection, 468 cholesterol, colestyramine, kidney function, 433 chronic allograft nephropathy, kidney transplantation, 618 - mycophenolic acid 2 morpholinoethyl ester, 628 chronic glomerulonephritis, angiotensin, proteinuria, 512 chronic kidney disease, 459 - s adenosylmethionine, hyperhomocysteinemia, 519 - anemia, heart left ventricle hypertrophy, hemoglobin, recombinant erythropoietin, 432 - anemia, recombinant erythropoietin, 448 - artery intima, cardiovascular risk, carotid artery, 523 - biological marker, 456 - body mass, 524 - bone metabolism, dialysis, kidney disease, 440 - cardiovascular disease, 457 553 - computer system, general practice, 444 - drug use, heart muscle ischemia, 445 - kidney donor, kidney transplantation, 442 - pressoreceptor reflex, vasomotor reflex, 460 - vasculotropin, 493 chronic kidney failure, 600 - amlodipine, blood pressure regulation, hypertension, quinapril, 466 - anemia, heart muscle ischemia, 436 - cellular immunity, continuous ambulatory peritoneal dialysis, innate immunity, 584 - essential hypertension, 5, 10 methylenetetrahydrofolate reductase FADH2 ; , nephrosclerosis, 521 - gene therapy, recombinant erythropoietin, 450 - hemodialysis, urea, 578 chronic liver disease, erectile dysfunction, protein malnutrition, 724 chyluria, surgical approach, surgical technique, 478 cigarette smoking, insulin dependent diabetes mellitus, kidney function, non insulin dependent diabetes mellitus, 491 cinacalcet, secondary hyperparathyroidism, 560 ciprofloxacin, cefotaxime, imipenem, recurrent infection, Salmonella enterica, salmonellosis, 590 circumcision, 798 cisplatin, magnesium, magnesium depletion, nephrotoxicity, 638 citrate sodium, citric acid, hemodialysis, 438 citric acid, acute kidney failure, anticoagulation, hemofiltration, 462 - citrate sodium, hemodialysis, 438 - hemofiltration, nutrition, 439 clusterin, antisense oligonucleotide, prostate cancer, 771 Section 28 vol 66.2.
Examples include quinapril hydrochloride, marketed by parke-davis under the accupril.
What are the side effects of quinapril
Beta-carotene + vitamin e beta-carotene 7 mg dl-î ± -tocopherol acetate 25 mg; softgels, orange, 2 oval, prophylactic appliance in stress exposure and in supplementation of poor green and yellow fruits and vegetable diet especially in winter and in environment polluted area and aceon.
Publications a drug price the Average Wholesale Price or "AWP" ; that is deliberately set far above the prices that their drugs are available in the marketplace. The AWPs for these drugs are deliberately false and fictitious and created solely to cause Plaintiffs and the Class members to overpay for drugs. Because all drugs administered under Medicare Part B and brand name drugs administered outside of the Medicare context are priced based on the published AWPs, the Defendant Drug Manufacturers inflate AWP reimbursement rates to enable providers and others to make secret profits through overcharges to patients and their insurers. This, in turn, incentivizes the providers to sell and administer the drugs with the most inflated AWPs, resulting in increased market share and profit for the Defendant Drug.
The 30th June 04 saw the close of the first year that Helping Hands in Nowra has been running under the auspice of Schizophrenia Fellowship. Helping Hands is a volunteer service based in Nowra that offers volunteer support to people with mental illness. It was established in 2001 by the Area Health Service AHS ; as a pilot project. The program trains volunteers, who are then linked with individual consumers from the Nowra and Ulladulla area to provide support in a range of activities such as shopping, social and recreational activities, education and training opportunities, etc. The volunteers also organise a range of group activities including sporting, recreational and education opportunities. The program is run by a coordinator with some administrative assistance. Sometimes the volunteers are the only people with whom the consumers have regular contact with. Volunteers also provide a vital link between the consumers and the community health services. The Helping Hands program initiates various activities to reduce the stigma of mental illness in the local community. Since the Fellowship took over the management of Helping Hands it has grown and developed. The number of volunteers has increased, as has the number of consumers who have links with volunteers. The profile of Helping Hands has been increased through media, talks and networking with health professionals and other non-government and government agencies. The program has been extended to consumers referred from agencies other than the mental health service.
Comparative evidence for health-related quality of life derives from 9 studies 22, 23, 25, ; . All.
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